I'm not clear on what exactly Methylation therapy is, but my understanding is that our bodies have toxins that need to be eradicated but are missing the proper nutrients to do this.  It is the toxin level that depletes the body's immuneⁱ system permitting pathogens to infiltrate it. 

A man named Rich Van Konynenburg has written extensively on this topic.  He too is a PhD who has a simple protocol. There are extensive postings by him on the ProHealth web site.  

Here is link to one thread which includes the testing lab he recommends in NJ.  Also, for those in Europe, a google search for Vitamin Diagnostics Lab will bring up sites that list world-wide testing labs.  

http://www.prohealth.com/fibromyalgia/blog/boardDetail.cfm?id=1323644 

Your doctor’s office will need to contact the lab to order the blood test kit (it’s just one or two vials), and they will send the kit to your doctor’s office.  One of the doctors at the lab called my doctor with a personalized treatment plan based on my results. Here’s the contact info:

From what I remember, it cost $200-$300, and my insurance did not cover it.   

Hope this helps. 

Methylation, to a chemist, is the adding of a methyl group (CH3) to a molecule. Biochemically, there is an apparatus involving vitamin B12, folateⁱ, and various other molecules, which gets used to methylate biomolecules. Of these, B12 is probably the most critical, since it's the essential catalyst for rejuvenating folate. The idea behind 'methylation therapy' is that people have some sort of defect in their methylation cycle. I'm not quite sure what it is, since I never had the patience to read that far; but I gather that it's one of a number of genetic differences, for which the 'methylation therapy' people offer tests. The idea that it might be a genetic defect doesn't make sense to me, since a genetic defect would have been obvious from childhood, whereas most of our diseasesⁱ are acquired. What does make sense to me is Stratton's claim that in porphyriaⁱ, antibodies get made to porphyrins, and some of them cross-react with B12 and diminish the usable amount of B12; thus his recommendation of high-dose B12 supplementation.

The bit on B12, porphyriaⁱ, and antibodies comes from one of the Stratton patents. The patent language is rather terse, so it's not clear what experiments may have been done to back up that claim. But antibody cross-reactions happen a fair bit; antibodies are never perfectly specific.

 

   When you get measured your B12 levels it may be reported as within normal limits, but this doesn't mean that your cells can easily use it. Infact ıf you are cpnⁱ infected and if you have huge amounths of antibodies, your B12 would be covered by these antibodies and your cells could't use it anymore. But there are ways for measuring your real B12 status.

 1)You can look at your homosistein level( a neurotoxin and cardiotoxin also-it give damage to connective tissue).It's level is elevated during B12,folic asit or pyridoxin deficiency. B12 and folic asit act as cofactor for conversion of homocystein to methionin, and pyridoxin act as cofactor for conversion of it to cystein.

  If homosistein is normal that is not certainly mean that your B12 is normal, because folic asit and pyridoxin might have normalized it even if you are B12 deficient. So you need a more reliable test

 2)You can look at your methyl malonate levels which is a better indicator of  B12 deficiency. B12 convert methyl malonyl coA to succinyl coA, so if you have high metil malonate levels that means you are B12 deficient with %99 possiblity.

  YILMAZ