Okay, it has been suggested by someone that NACⁱ may in fact not be doing what it supposed to be doing, namely, replacing amoxicillinⁱ in the CAPⁱ.  The role for both is to eradicate the EBⁱ stage of Cpnⁱ, which is intra-cellular, floating around in the blood, plasma, and other fluids in the body, seeking to find a cell to infect and become an RB.  Both NAC and Amoxicillin come along and interrupt the di-sulphide bonds that hold an EB closed when floating around.  When it opens, it begins to need food and starves.  That how we kill an EB.

So, the question is, is NAC doing this effectively?  Is it possible that we're counteracting it somehow, since NAC is a precursor to Glutathione, are we maybe generating reverse NAC to bring levels down, the same way our body will regulate other fluid and proteins in it?  That is a possibility I can neither check or eliminated, but I'm hoping someone will have information that can do that.