Initial and following blood work is not just a matter of Cpn related indicators, but also relevant to your particular history and case, as determined by your doctor. Suggestions drawn from experts treating Cpn in a variety of conditions include the following.
Initial blood work can be obtained for the following tests:
- CBC & Differential
- Liver function tests
- Uric acid
- Serum iron studies (typically depleted by Cpn: low iron levels are more diagnostic, and are not necessariy indicators to supplement, which may actually increase Cpn infection-- see references below).
- Red blood cell ALA dehydratase
- Red blood cell PBG deaminase
- Vitamin B-12 level
- Homocysteine levels
- Serum methymalonate level.
- Vitamin D levels
- Thyroid panels (standard plus free T4, free T3, revers T3) [Endocrine disturbances common in Cpn and associated diseases]
- 24-hour urine and 24-hour stool specimens for porphyrins
Dr Stratton has noted relative to porphyrins:
Homocystine levels are elevated with B12 and folate deficiency, but can be reduced by folate alone. On the other hand, serum methyl malonate levels are elevated in B12 deficiency and are not changed by folate. Therefore, serum methyl malonate levels are the best indicator of B12 deficiency.
Another indicator, according to Dr. Stratton, is high hemoglobin and high hematocrit.
Dr. Powell notes:
I also tst DHEA-S and free testosteron in perimenopausal females. Both increase nitric oxide levels, which kills Cpn. No point heading into treatment with low androgens.
Regular Followup Tests
- CBC & Differential
- Liver function tests (especially important when using medications such as INH or Rifamcin which can have liver toxicity, and because die-off of liver cells infected with Cpn can affect liver function)
- Vitamin D levels (if supplementing deficiency)
- Thyroid panels (standard plus free T4, free T3, reverse T3) (if supplementing deficiency)
- Others as determined by doctor relevant to your particular condition.
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