27 Apr 2018

Lyme disease


Hi, I have just been diagnosed with Lyme disease and am wondering if the CAP treatment for Cpn will also do th trick for Lyme?  I have been on the abx treatment for Cpn coming up to 11 months now, just hoping I won'y have to add anything further due to the lyme diagnosis.Thanks :)


It's the answer I have also been searching for. The CAP is dsigned to attack cpn at all stages of development. It is based on research by Drs. Stratton and Wheldon. Surely people have been helped by it. It has slowed the progression of my neurological symptom but neither stopped nor reversed them. Whether borrelia, babesia and bartonella have exactly the same life cycle vulnerabilities as cpn has not, I believe, been studied and may be the reason that I am not one of the success stories.My diagnosis is ppms. Is that yours as well?

PPMS-misdiagnosed 2001-diagnosed 2006. Probably caught cpn in birth canal but it didn't pass BBB until my 40s. Minocycline 7 mos.- resulting bronchitis 5 months.Go to private m.d. out-of-plan. Wheldon CAP 3/2/07 Stopped 12/12; resumed 12/13

Don't know about Melly, but I have chlamydia p. and then all of the above - Lyme borrelia, babesia etc. all of the co-infections. I am on CAP since end of June...so don't know more than that. I didn't even know I was sick! I twitch less, less muscle/joint pain is all. But weird things in my abdomen now. No MS diagnosis so far but they said I had numerous scattered white matter foci in my brain.... so who knows...I have been to a Lyme doc and he treated with doxy. If you have heavy metal in your body it will let the infection be worse..all I know.Linda

I was diagnosed with rrms...had 5 relapses last year which left me unable to walk without aid.  Since starting the CAP I have not had a relapse so somethings working. I am just not sure whether this is enough for lyme also or whether I should do something else :/

The combination is effective for Bb and it is a delicate balance in going fast enough to make progress over time and treating so agressively with individuals with neurological involvment that it causes increased neuro symptoms which can scare them off or possibly kill of to many cells at one time for the body to heal all of them quickly enought to prevent the individual for becoming scared and stopping.  Also the concentration of the secondary ab is sometime higher in treating Bb.   Problem with the Bb treatment by  the so called literate practitioner is they underestimate the involvement or tenacity of the C.Pn. infections in the mix.  The capacity of the C.Pn. to inhibit natural apoptosis of the cell allows the mix of infections that are intracellular in nature to survive as they shift shape and form in response to abx tx.For myself once I was stable on my dosage I increased my Roxi to 300 twice a day to be a bit more agressive in treatment.    I had not progress to neurological symptoms in any significant way nor had a dx of ms  some folks said that I had a situation like they did early in their disease I think it all may be a continuum and various factors contribut to ones progression and symptomatology hence why everyone looks and displays different symptoms.Not sure that is at all helpful but I am not willing to have a long discussion of this as I am too busy being back in a productive life thanks to the information I got being here in this community over the past 7 years this past Spring.    Louise

  • CAP(TiniOnly): 06/07-02/09 for CFS
  • MethylationProtocolSupplements: Started08/08
  • Intermtnt CAP: 02/09-02/10
  • Full MethylProtocol & LDN 02/09
  • Off CAP: 02/10, cont LDN & MethlyProtocol support

Melly, I agree with Supaguy!How were you diagnosed with lyme and who by?................................Sarah

Completed Stratton/Wheldon regime for aggressive secondary progressive MS in June 2007, after four years, three of which intermittent.   Still improving bit by bit and no relapses since finishing treatment.