Abdominal Aortic Aneurism

This didn't seem to be in our list of diseases, but Dianna, my new, tireless googler of all things chlamydial, found it: http://www.smokershistory.com/cpaaa.htm They may have an ax to grind, but the number of papers cited seems pretty convincing.

When I link that information with David's http://www.davidwheldon.co.uk/chl_vasc.html (he mentions aneurism), where he describes the wide pulse pressure that develops with C.Pn., and with Peckerman's tilt-table results that show CFS is characterised by diastolic dystrophy, it sounds even more likely.

I know (but can't remember from where) that AAA's are most likely to occur at a point about one-half wavelength above the bifurcation of the aorta (where it splits to form the femoral arteries.) That's where the "echo" bouncing back from the split meets and reinforces the pulse coming down from the heart. It seems plausible that the stiffened aorta would make a sharper pulse and echo, and also can't "soak up" the shock wave at that point the way it should.

Scary.

 

Ron 

If this list doesn't convince you of the connection between Cpn and heart/vascular disease, I don't know what will. Thanks for this great find, Ron. We'll integrate some of these abstracts into our research section.

I'll be interested to hear David's comments on this, since it is the vascular component he treated in himself with the Cpn protocol. 

On Wheldon/Stratton protocol for Cpn in CFS/FMS since December 2004.

 

CAP for Cpn 11/04. Dx: 25+yrs CFS & FMS. Currently: 250 aithromycin mwf, doxycycline 100mg BID, restarted Tini pulses; Vit D2000 units, T4 & T3, 6mg Iodoral

My abdominal aorta has pulsed so hard for years i can see it beating through my skin.

minocycline, azithromycine, metronidazole 2007-2009, chelation for lead poisoning, muscle pain, insomnia, interstitial cystitis (almost well), sinus, dry eyes, stiff neck, veins, hypothyroid, TMJ, hip joints (no longer hurt)

A wishbone pattern of pain has radiated down my abdomin and split down my legs for years. I started antibiotics a few days ago. I have broken out in a rash over the wishbone. Maybe endotoxins from dying Cpn are moving to the surface.

minocycline, azithromycine, metronidazole 2007-2009, chelation for lead poisoning, muscle pain, insomnia, interstitial cystitis (almost well), sinus, dry eyes, stiff neck, veins, hypothyroid, TMJ, hip joints (no longer hurt)

D W

Food for thought here. When you see the aorta of someone with bad arteriopathy you just wonder how they survived so long; it's rigid, brittle and calcified. The pulse must go down it like a hammer-blow. Yet the stiffening begins surprisingly early in life, and is associated with raised cpn antibodies. The pathology is worst in the areas of greatest stress, as you say, Ron.

 

Just a minor note, but not as off-topic as might be supposed. It's sometimes the smaller and less obvious pieces of evidence which set you thinking the hardest; possibly because they are unanticipated. Like many people in mid life I had a progressive deterioration in eye refractive function; this had started in about 1985 and slowly and steadily got worse. Two-yearly visits to the optician resulted in prescriptions for ever-stronger lenses. After taking antibiotics and supplements, I found that this deterioration of refraction stopped. On my last visit to the optician he said 'That's amazing: your refraction is improving.' It's true; I could read lines on the chart I couldn't read three years ago. So unexpected. But if this resolution takes place in tissues all over the body, what might that mean? A lot of events we put down to aging might be due to chronic infection.

 

David

D W - [Myalgia and hypertension (typically 155/95.) Began (2003) taking doxycycline and macrolide and later adding metronidazole. No medication now. Morning BP typically 110/75]

David, the same is true with me. I still wear bifocals I bought one year before the diagnosis in Nov, 2002. I was on daily supplements and Lipitor till Feb, 2005.I don't use them for reading anymore.If I decide to buy a new pair I will get glasses for myopia only.

Cured of multiple sclerosis, stopped the Wheldon's protocol in Nov,2008. Use only LDN.

D W

Ice, that's very funny! It's not the sufferers who are myopic.
D W - [Myalgia and hypertension (typically 155/95.) Began (2003) taking doxycycline and macrolide and later adding metronidazole. No medication now. Morning BP typically 110/75]

Very interesting. The MS doc who swore up and down that I didn't have MS ultimately told me he thought I had a "blood flow problem". All along it was Cpn. Too bad he left the center where I saw him. I would love to tell him it he was "close but no cigar". I no longer feel short of breath when I take a long drink of water and my heart doesn't pound afterward. I think I was circling the drain and would have died an early death if I hadn't found this treatment. Thank you Dr. Stratton, I could kiss you. Raven

Feeling 98% well-going for 100. Very low test for Cpn. CAP since 8-05 for Cpn/Mycoplasma P.,Lyme, Bartonella, Mold exposure,NAC,BHRT, MethyB12 FIR Sauna. 1-18-11 begin new treatment plan with naturopath

You know raven....I have noticed also the 'not being out of breath' when taking a long drink of water!  I also noticed that splashing cold water on my face in the a.m. doesn't shock me anymore or set my ears to ringing.

 

Geez...It makes me wonder...just how long HAVE I been this way?  Probably since before the ms dx.

:)

Wheldon Protocol for rrms since Oct '05.  Added LDN 4.5mg qhs Oct '07.  All supp's.  Positive IGG's for Lyme Disease,Babesia, & Erlichiosis Sept. 2008.  Currently:  Mepron 750mg bid and Azithromycin 250mg qd for Babesia.

Interesting trend here -- my wife has just moved my "start date" back about 8 years, based on some of the subtler symptoms that people here have reported. I wonder how I'll recognize normal when I get there.

And I wonder how many of us will start to see things we didn't associate with this disease process as a part of the whole CPn picture?

 

Ron 

Ron

On CAP for CFS starting 01/06 (NE Ohio, USA)

Began rifampin trial 1/14/09

Currently: on intermittent


Abdominal Aortic Aneurysm: Causes, Symptoms and Diagnosis
posted on: Sat, october 2010
posted by : manuheart123
An abdominal aortic aneurysm is said to have occurred, when the large blood vessel that carries blood away from the heart and supplies to the abdomen, pelvis and legs, dilates or bulges. This localised dilatation of the abdominal aorta exceeds the normal diameter by more than 50 percent. Aneurysms also occur in other areas of the aorta, but the abdomen is the most common site.